Fortell noe helt uinteressant!

[Gjest LightBulb]

Vel, jeg foreslår at dere heller drikker ti kopper grønn te fremfor å hive i dere nervegift. Noen blir faktisk demente av alkohol.

Hvem er du? hvor er jeg? Hva gjør jeg her? Nuvel, får drikke litt mer..

[Rumle]

[Giacomo_P]

Odisi, kanskje du sovner letter hvis du tar én av de sauene og legger i sengen, så det bliver godt og varmt?

[*appifany*]

Hvem er du? hvor er jeg? Hva gjør jeg her? Nuvel, får drikke litt mer..

Minner meg litt om den barnesangen?! Hvor er du? Her er jeg! Synger du? Det gjør jeg! Heisann og hoppsann og fallerallera(osv, osv)

Vi har nå stadfestet at den ble skrevet i fylla!!

[Gjest odisi73]

Jeg vurderer å fange en i lufta

[Giacomo_P]

Jeg tror jeg tar meg et glass Metanol

Jeg tror jeg tar meg et glass metadon....

[*appifany*]

[Gjest LightBulb]

Minner meg litt om den barnesangen?! Hvor er du? Her er jeg! Synger du? Det gjør jeg! Heisann og hoppsann og fallerallera(osv, osv)

Vi har nå stadfestet at den ble skrevet i fylla!!

av en som har drukket seg dement!

[Rumle]

Endret 5. desember 2011 av Rumle

[*appifany*]

av en som har drukket seg dement!

Tror de fleste barnesanger blir skrvet i den tilstanden!

[AnonymBruker]

Wernicke-Korsakoff?

Da skal du virkelig jobbe med konsumet over lang, lang tid. De fleste dør av andre ting lenge før de udvikler slikt.

The effect of alcohol on the nervous system is even more pronounced among alcohol-dependent individuals. Chronic high doses cause peripheral neuropathy in 10% of alcoholics: similar to diabetes, patients experience bilateral limb numbness, tingling, and paresthesias, all of which are more pronounced distally. Approximately 1% of alcoholics develop cerebellar degeneration or atrophy. This is a syndrome of progressive unsteady stance and gait often accompanied by mild nystagmus; neuroimaging studies reveal atrophy of the cerebellar vermis. Fortunately, very few alcoholics (perhaps as few as 1 in 500 for the full syndrome) develop Wernicke's (ophthalmoparesis, ataxia, and encephalopathy) and Korsakoff's (retrograde and anterograde amnesia) syndromes, although a higher proportion have one or more neuropathologic findings related to these syndromes. These occur as the result of low levels of thiamine, especially in predisposed individuals, e.g., those with transketolase deficiency. Alcoholics can manifest cognitive problems and temporary memory impairment lasting for weeks to months after drinking very heavily for days or weeks. Brain atrophy, evident as ventricular enlargement and widened cortical sulci on MRI and CT scans, occurs in 50% of chronic alcoholics; these changes are usually reversible if abstinence is maintained. There is no single alcoholic dementia syndrome; rather, this label is used to describe patients who have apparently irreversible cognitive changes (possibly from diverse causes) in the context of chronic alcoholism.

[Rumle]

[Gjest odisi73]

Nå er det ene øyet mitt igjen håper det andre også detter igjen snart

[*appifany*]

Er ikke lov med copy-paste uten å henvise til link.

[Giacomo_P]

Tror de fleste barnesanger blir skrvet i den tilstanden!

Ikke Margrethe Munthes barnesanger. De er nok skrevet i patologisk avhold, for å skremme alle under 50.

[AnonymBruker]

Er ikke lov med copy-paste uten å henvise til link.

Gjerne det, men tror nok ikke du har tilgang.

http://www.accessmedicine.ca/content.aspx?aID=9149630

[Gjest LightBulb]

The effect of alcohol on the nervous system is even more pronounced among alcohol-dependent individuals. Chronic high doses cause peripheral neuropathy in 10% of alcoholics: similar to diabetes, patients experience bilateral limb numbness, tingling, and paresthesias, all of which are more pronounced distally. Approximately 1% of alcoholics develop cerebellar degeneration or atrophy. This is a syndrome of progressive unsteady stance and gait often accompanied by mild nystagmus; neuroimaging studies reveal atrophy of the cerebellar vermis. Fortunately, very few alcoholics (perhaps as few as 1 in 500 for the full syndrome) develop Wernicke's (ophthalmoparesis, ataxia, and encephalopathy) and Korsakoff's (retrograde and anterograde amnesia) syndromes, although a higher proportion have one or more neuropathologic findings related to these syndromes. These occur as the result of low levels of thiamine, especially in predisposed individuals, e.g., those with transketolase deficiency. Alcoholics can manifest cognitive problems and temporary memory impairment lasting for weeks to months after drinking very heavily for days or weeks. Brain atrophy, evident as ventricular enlargement and widened cortical sulci on MRI and CT scans, occurs in 50% of chronic alcoholics; these changes are usually reversible if abstinence is maintained. There is no single alcoholic dementia syndrome; rather, this label is used to describe patients who have apparently irreversible cognitive changes (possibly from diverse causes) in the context of chronic alcoholism.

aah, aha... jasså..

I don't give a rats ass!

Legg deg nå, du ødelegger fyllafesten! Hæla i taket og tenna i tapeten!

[*appifany*]

Gjerne det, men tror nok ikke du har tilgang.

http://www.accessmedicine.ca/content.aspx?aID=9149630

Da tror jeg også du burde stoppe...

[AnonymBruker]

Da tror jeg også du burde stoppe...

Hva mener du?

[Rumle]

Jaja, nå går jeg jammen og legger meg ... natta alle sammen!